Search results for "Hippocampal synaptosome"

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Neurosteroid modulation of the presynaptic NMDA receptors regulating hippocampal noradrenaline release in normal rats and those exposed prenatally to…

2003

Abstract Prenatal exposure to diazepam (DZ), a positive allosteric modulator of the γ-aminobutyric acidA (GABAA) receptor complex, exerts profound effects that become more evident during puberty and in many cases are sex-specific, suggesting that such exposure interferes with the activity of steroid hormones. Apart from their well known effects on the genome, the reduced metabolites of many steroid hormones also interact directly with membrane receptors, including those for N-methyl- d -aspartate (NMDA). In this study, we compared the effects of several neurosteroids on NMDA receptors from normal rats and those exposed in utero to DZ (1.25 mg/kg per day) from the 14th through the 20th day o…

MalePregnenolone sulfatemedicine.medical_specialtyReceptor complexNeuroactive steroidAllosteric modulatorGlycinePharmacologyHippocampusReceptors N-Methyl-D-AspartateNorepinephrineCellular and Molecular Neurosciencechemistry.chemical_compoundPregnancyInternal medicineNeurosteroidmedicinepregnenolone sulphateAnimalsRats WistarReceptorDiazepamGABAA receptorHippocampal synaptosomesCell BiologyRatsEndocrinologyNMDA/GLY-mediated [3H]NA releasechemistryPregnenolonePrenatal Exposure Delayed EffectsSettore BIO/14 - FarmacologiaNMDA receptorFemalePregnenolone sulfateSynaptosomesHormoneNeurochemistry International
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Presynaptic effects of anandamide and WIN55,212-2 on glutamatergic nerve endings isolated from rat hippocampus

2006

We examined the effects of the endocannabinoide-anandamide (AEA), the synthetic cannabinoid, WIN55,212-2, and the active phorbol ester, 4-beta-phorbol 12-myristate 13-acetate (4-beta-PMA), on the release of [(3)H]d-Aspartate ([(3)H]d-ASP) from rat hippocampal synaptosomes. Release was evoked with three different stimuli: (1) KCl-induced membrane depolarization, which activates voltage-dependent Ca(2+) channels and causes limited neurotransmitter exocytosis, presumably from ready-releasable vesicles docked in the active zone; (2) exposure to the Ca(2+) ionophore-A23187, which causes more extensive transmitter release, presumably from intracellular reserve vesicles; and (3) K(+) channel block…

MaleSettore BIO/14 - FARMACOLOGIAPolyunsaturated AlkamideshippocampusMorpholinesmedicine.medical_treatmentPresynaptic TerminalsArachidonic AcidsNaphthalenesExocytosisCellular and Molecular Neurosciencechemistry.chemical_compoundGlutamatesglutamate releasemedicineAnimalsanandamideActive zoneRats WistarNeurotransmitterCannabinoidCalcimycinProtein kinase CSynaptosomeArachidonic AcidChemistrysynaptosomesDepolarizationCell BiologyAnandamideHippocampal synaptosomeCalcium Channel BlockersBenzoxazinesRatsBiochemistryBiophysicsTetradecanoylphorbol AcetateCannabinoidCapsaicinEndocannabinoidsNeurochemistry International
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